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The addition of exogenous GSH was not only able to restore its intracellular levels almost up to those found in uninfected cells, but also to completely inhibit the HSV-1 replication (Palamara et al., 1995)
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4a, b), which was not affected by the inclusion of TNF in the media (Fig
Mimics of the dimerization domain of hepatocyte growth factor exhibit anti-Met and anticancer activity
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