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Right now Im doing some research on an exploratory piece around ACA exchange growth and economic effects of that growth in 2024, and this trend is pretty notable: Compared to the same time in 2022, as folks get bumped off Medicaid redeterminations theyre finding themselves eligible for exchange plans based on the expanded subsidies in place through 2025 (Link) 33 percent more people have enrolled in marketplace plans in states that use HealthCare.gov. Floridas Live Healthy state proposals held some interesting healthcare provisions: Along with the above, Beckers pointed out a few other high level provisions that the state is targeting in the package: Alleviating the healthcare workforce problem Making it easier for providers to practice in critical need areas Addressing ER and acute care admissions & boosting funding for programs to divert these volumes (e.g., the above hospital at home initiative to get Florida Medicaid funding for the program) Money for hospitals for labor & delivery programs and GME programs for highly specialized tertiary care Creation of an innovation council to learn of new solutions for labor and care delivery innovation Autism and IDD Care Growth: Demand for autism and intellectual and developmental disabilities (IDD) care is at an all-time high, according to a CentralReach report

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Research has examined its role in: GHRH receptor binding, Gs/adenylate cyclase activation, and cAMP/PKA signalling pathway studies Physiological pulsatile GH secretion modelling and somatotroph biology investigations GH and IGF-1 axis regulation and downstream anabolic signalling research DPP-4 resistance profiling and GHRH analogue structural pharmacology studies Somatotroph proliferation, GH mRNA transcription, and pituitary reserve research GH pulsatility architecture pulse amplitude, frequency, trough levels, and inter-pulse intervals Somatostatin feedback dynamics and GHRH/somatostatin interplay modelling Metabolic pathway research lipid oxidation, glucose homeostasis, and anabolic signalling GH axis ageing, somatopause, and GHRH pulse amplitude decline studies Dual-pathway synergy research when combined with GHS-R1a agonists such as Ipamorelin Comparative GHRH analogue pharmacology Sermorelin, Tesamorelin, and CJC-1295 DAC CJC-1295 Without DAC and Pituitary Somatotroph Biology Research in GHRH knockout mice using the CJC-1295 backbone demonstrated that administration caused an increase in total pituitary RNA and GH mRNA, suggesting that proliferation of somatotroph cells had occurred as confirmed by immunohistochemistry establishing that the tetrasubstituted GHRH scaffold acts not only on acute GH release but also on transcriptional upregulation of GH gene expression and pituitary somatotroph reserve, findings directly relevant to the no-DAC variants receptor biology