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This mechanism establishes OLA as a promising anti-fibrotic therapeutic candidate ( Mitochondria-targeted antioxidants or CYP2E1 inhibition prevents this toxicity, highlighting CcO dysfunction as a key pathological mechanism ( Daphnia magna through its reactive metabolite NAPQI

It is studied for potential roles in tissue repair, inflammation regulation, and gastrointestinal protection, but remains an investigational compound without approved medical use

Never inject water directly onto the lyophilized powder