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Samples were then analyzed by high-performance liquid chromatography/mass spectrometry in order to determine drug concentrations and calculate negative controls samples containing no liver microsomes [2]

Integrated multi-omics and experimental validation reveal FDX1/LIAS-mediated cuproptosis as a potential driver of diabetic kidney disease

Bone marrow stromal cell-derived exosomal circular RNA improves diabetic foot ulcer wound healing by activating the nuclear factor erythroid 2-related factor 2 pathway and inhibiting ferroptosis

The key is duration-appropriate exposure: colder temperatures require shorter times

No clinical trials include TB-500 cycles longer than 2 weeks [15, 16]