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Upon ligand binding, TR II phosphorylates and activates TR I, forming a heteromeric receptor complex that initiates downstream signaling through both SMAD-dependent and SMAD-independent pathways, such as ERK, p38, JNK, and PI3K-Akt cascades [33, 36, 39]
It has also been suggested that oxidative modifications of DNA and RNA may affect the heredity of BD (Brown et al
Further studies are required to clarify the structural and functional bases of this selective catalysis
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