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Switching mutant Sod1 mice from a normal to a ketogenic diet, or giving them caprylic triglyceride, which is metabolized to ketone bodies, restored their mitochondrial ATP production and was accompanied by reduced neuronal loss, delayed onset of motor dysfunction, and enhanced survival [193, 194]
Beyond week 6, results plateau unless caloric deficit is maintained or injection frequency increases (some protocols move to twice-weekly dosing after week 8)
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